Professor Deborah Ferrington, University of Minnesota.
1:00pm - 2:00pm /
Wednesday 18th September 2019
/ Venue: William Henry Duncan Apex Building
- 0151 794 9003
- Brenda Smith
- Suitable for: Staff and students
- Admission: Free to staff and students
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Deborah Ferrington, Professor and Director of Research, Department of Ophthalmology and Visual Neurosciences, University of Minnesota
‘Matching Therapy to the Defect: Targeting the Mitochondria to Treat Age-Related Macular Degeneration.’
Dr Ferrington’s research is focused on investigating the molecular changes that occur with retinal aging and age-related macular degeneration (AMD), the number one cause of blindness among adults in the developed world. The experimental approach to investigate AMD pathogenesis has been to study changes in cellular composition and function in human donor eyes at progressive stages of AMD using a combination of proteomic and molecular biology techniques, as well as performing functional testing on RPE cultures. Results from multiple analyses led to the unique observation that the mitochondria in the retinal pigment epithelium (RPE) are negatively impacted by the disease and thus, mitochondria are a potential molecular target for therapy.
Host: Luminita Paraoan
‘Matching Therapy to the Defect: Targeting the Mitochondria to Treat Age-Related Macular Degeneration.’
Dr Ferrington’s research is focused on investigating the molecular changes that occur with retinal aging and age-related macular degeneration (AMD), the number one cause of blindness among adults in the developed world. The experimental approach to investigate AMD pathogenesis has been to study changes in cellular composition and function in human donor eyes at progressive stages of AMD using a combination of proteomic and molecular biology techniques, as well as performing functional testing on RPE cultures. Results from multiple analyses led to the unique observation that the mitochondria in the retinal pigment epithelium (RPE) are negatively impacted by the disease and thus, mitochondria are a potential molecular target for therapy.
Host: Luminita Paraoan